caspase 1 activity assay kit (Beyotime)
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caspase 1 activity assay kit
Caspase 1 Activity Assay Kit, supplied by Beyotime, used in various techniques. Bioz Stars score: 96/100, based on 517 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/caspase+1+activity+assay+kit/Caspase+1+Activity+Assay+Kit/pm42815265-151-1-6
Average 96 stars, based on 517 article reviews
Caspase 1 Activity Assay Kit, supplied by Beyotime, used in various techniques. Bioz Stars score: 96/100, based on 517 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/caspase+1+activity+assay+kit/Caspase+1+Activity+Assay+Kit/pm42815265-151-1-6
Average 96 stars, based on 517 article reviews
caspase 1 activity assay kit - by Bioz Stars,
2026-10
96/100 stars
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Activity Assay:Article Title: Overconsumption of fructose aggravates acute GVHD by inducing gut dysbiosis and promoting macrophage-mediated inflammatory response. Article Snippet: .. Article Title: Ag 2 Se quantum dots neurotoxicity: mitochondrial stress drives ferroptosis-dependent microglial inflammation. Article Snippet: .. 2.18 Caspase-1 activity assay Caspase-1 activity was measured using the Article Title: Embryonic exposure to bisphenol A glycidyl methacrylate induces craniofacial cartilage malformation via NLR signaling pathway in zebrafish. Article Snippet: Apocynin (APO, CAS: 498–02–2) was purchased from Santa Cruz Biotechnology (St Louis, USA). .. Article Title: The novel piperlongumine-ligustrazine derivative PL-Lig-11 inhibits VSMC phenotypic switching and intimal hyperplasia via the PDGFRα/β-RhoGDI3-NLRP3 inflammasome axis. Article Snippet: Neointimal hyperplasia driven by vascular smooth muscle cell (VSMC) phenotypic switching is a key pathological process in cardiovascular diseases.. This study investigated the effects and mechanisms of PL-Lig-11, a novel derivative combining piperlongumine (PL) and ligustrazine (Lig), on VSMC phenotypic switching and intimal hyperplasia.. PL-Lig-11 significantly suppressed PDGF-BB-induced VSMC proliferation, migration, and phenotypic transformation, and attenuated intimal thickening in rat carotid arteries, showing superior efficacy to the parent compounds. Article Title: Infectious bronchitis virus accessory protein 3a induces renal inflammatory injury by activating the NLRP3 inflammasome via ER calcium mobilization and mitochondrial ROS production. Article Snippet: Lipofectamine 2000 Transfection Reagent (11668019) was purchased from Invitrogen. .. Article Title: Ningxintong and its active component calycosin stabilize vulnerable atherosclerotic plaques by targeting SIRT7 and inhibiting pyroptosis. Article Snippet: Objective: To investigate the regulatory effect of Ningxintong (NXT) on monocyte/macrophage pyroptosis in atherosclerosis (AS), elucidate its molecular mechanisms, and identify its key active components.. Methods: The expression of inflammationand pyroptosis-related molecules was detected in peripheral blood monocytes from patients with AS.. An atherosclerotic vulnerable plaque (AS-VP) mouse model was established to evaluate the effects of NXT on blood lipid levels, plaque formation, and stability. Article Title: High‐Throughput Screening Reveals That CeeNU Acts as a New NLRP3 Inflammasome Inhibitor Article Snippet: Anti‐mouse IL‐1β, Human IL‐1β, IL‐6, and TNF‐α ELISA kits (CEK1788, CEK1731, CEK1785, CEK1783) were from Bioworld (USA). .. Cytotoxicity (LDH) assay kit ( C10007 ), Article Title: DS-10 mitigates cerebral ischemia-reperfusion injury in mice through suppressing caspase-1-mediated pyroptosis. Article Snippet: Ischemic stroke (IS) is an acute cerebrovascular event with substantial mortality and morbidity, yet effective therapies are lacking.. This study investigates the protective properties of DS-10, a decapeptide from Dunaliella salina, against cerebral ischemia-reperfusion injury (CIRI).. Using a mouse model of middle cerebral artery occlusion/reperfusion (MCAO/R) and oxygen-glucose deprivation/reoxygenation (OGD/R)-treated BV2 microglial cells, we evaluated the effects of DS-10. Transfection:Article Title: The novel piperlongumine-ligustrazine derivative PL-Lig-11 inhibits VSMC phenotypic switching and intimal hyperplasia via the PDGFRα/β-RhoGDI3-NLRP3 inflammasome axis. Article Snippet: Neointimal hyperplasia driven by vascular smooth muscle cell (VSMC) phenotypic switching is a key pathological process in cardiovascular diseases.. This study investigated the effects and mechanisms of PL-Lig-11, a novel derivative combining piperlongumine (PL) and ligustrazine (Lig), on VSMC phenotypic switching and intimal hyperplasia.. PL-Lig-11 significantly suppressed PDGF-BB-induced VSMC proliferation, migration, and phenotypic transformation, and attenuated intimal thickening in rat carotid arteries, showing superior efficacy to the parent compounds. SDS Page:Article Title: The novel piperlongumine-ligustrazine derivative PL-Lig-11 inhibits VSMC phenotypic switching and intimal hyperplasia via the PDGFRα/β-RhoGDI3-NLRP3 inflammasome axis. Article Snippet: Neointimal hyperplasia driven by vascular smooth muscle cell (VSMC) phenotypic switching is a key pathological process in cardiovascular diseases.. This study investigated the effects and mechanisms of PL-Lig-11, a novel derivative combining piperlongumine (PL) and ligustrazine (Lig), on VSMC phenotypic switching and intimal hyperplasia.. PL-Lig-11 significantly suppressed PDGF-BB-induced VSMC proliferation, migration, and phenotypic transformation, and attenuated intimal thickening in rat carotid arteries, showing superior efficacy to the parent compounds. Lysis:Article Title: The novel piperlongumine-ligustrazine derivative PL-Lig-11 inhibits VSMC phenotypic switching and intimal hyperplasia via the PDGFRα/β-RhoGDI3-NLRP3 inflammasome axis. Article Snippet: Neointimal hyperplasia driven by vascular smooth muscle cell (VSMC) phenotypic switching is a key pathological process in cardiovascular diseases.. This study investigated the effects and mechanisms of PL-Lig-11, a novel derivative combining piperlongumine (PL) and ligustrazine (Lig), on VSMC phenotypic switching and intimal hyperplasia.. PL-Lig-11 significantly suppressed PDGF-BB-induced VSMC proliferation, migration, and phenotypic transformation, and attenuated intimal thickening in rat carotid arteries, showing superior efficacy to the parent compounds. Article Title: Infectious bronchitis virus accessory protein 3a induces renal inflammatory injury by activating the NLRP3 inflammasome via ER calcium mobilization and mitochondrial ROS production. Article Snippet: Lipofectamine 2000 Transfection Reagent (11668019) was purchased from Invitrogen. .. Staining:Article Title: The novel piperlongumine-ligustrazine derivative PL-Lig-11 inhibits VSMC phenotypic switching and intimal hyperplasia via the PDGFRα/β-RhoGDI3-NLRP3 inflammasome axis. Article Snippet: Neointimal hyperplasia driven by vascular smooth muscle cell (VSMC) phenotypic switching is a key pathological process in cardiovascular diseases.. This study investigated the effects and mechanisms of PL-Lig-11, a novel derivative combining piperlongumine (PL) and ligustrazine (Lig), on VSMC phenotypic switching and intimal hyperplasia.. PL-Lig-11 significantly suppressed PDGF-BB-induced VSMC proliferation, migration, and phenotypic transformation, and attenuated intimal thickening in rat carotid arteries, showing superior efficacy to the parent compounds. Article Title: Infectious bronchitis virus accessory protein 3a induces renal inflammatory injury by activating the NLRP3 inflammasome via ER calcium mobilization and mitochondrial ROS production. Article Snippet: Lipofectamine 2000 Transfection Reagent (11668019) was purchased from Invitrogen. .. SYBR Green Assay:Article Title: The novel piperlongumine-ligustrazine derivative PL-Lig-11 inhibits VSMC phenotypic switching and intimal hyperplasia via the PDGFRα/β-RhoGDI3-NLRP3 inflammasome axis. Article Snippet: Neointimal hyperplasia driven by vascular smooth muscle cell (VSMC) phenotypic switching is a key pathological process in cardiovascular diseases.. This study investigated the effects and mechanisms of PL-Lig-11, a novel derivative combining piperlongumine (PL) and ligustrazine (Lig), on VSMC phenotypic switching and intimal hyperplasia.. PL-Lig-11 significantly suppressed PDGF-BB-induced VSMC proliferation, migration, and phenotypic transformation, and attenuated intimal thickening in rat carotid arteries, showing superior efficacy to the parent compounds. Real-time Polymerase Chain Reaction:Article Title: The novel piperlongumine-ligustrazine derivative PL-Lig-11 inhibits VSMC phenotypic switching and intimal hyperplasia via the PDGFRα/β-RhoGDI3-NLRP3 inflammasome axis. Article Snippet: Neointimal hyperplasia driven by vascular smooth muscle cell (VSMC) phenotypic switching is a key pathological process in cardiovascular diseases.. This study investigated the effects and mechanisms of PL-Lig-11, a novel derivative combining piperlongumine (PL) and ligustrazine (Lig), on VSMC phenotypic switching and intimal hyperplasia.. PL-Lig-11 significantly suppressed PDGF-BB-induced VSMC proliferation, migration, and phenotypic transformation, and attenuated intimal thickening in rat carotid arteries, showing superior efficacy to the parent compounds. Enzyme-linked Immunosorbent Assay:Article Title: The novel piperlongumine-ligustrazine derivative PL-Lig-11 inhibits VSMC phenotypic switching and intimal hyperplasia via the PDGFRα/β-RhoGDI3-NLRP3 inflammasome axis. Article Snippet: Neointimal hyperplasia driven by vascular smooth muscle cell (VSMC) phenotypic switching is a key pathological process in cardiovascular diseases.. This study investigated the effects and mechanisms of PL-Lig-11, a novel derivative combining piperlongumine (PL) and ligustrazine (Lig), on VSMC phenotypic switching and intimal hyperplasia.. PL-Lig-11 significantly suppressed PDGF-BB-induced VSMC proliferation, migration, and phenotypic transformation, and attenuated intimal thickening in rat carotid arteries, showing superior efficacy to the parent compounds. Immunostaining:Article Title: Infectious bronchitis virus accessory protein 3a induces renal inflammatory injury by activating the NLRP3 inflammasome via ER calcium mobilization and mitochondrial ROS production. Article Snippet: Lipofectamine 2000 Transfection Reagent (11668019) was purchased from Invitrogen. .. Blocking Assay:Article Title: Infectious bronchitis virus accessory protein 3a induces renal inflammatory injury by activating the NLRP3 inflammasome via ER calcium mobilization and mitochondrial ROS production. Article Snippet: Lipofectamine 2000 Transfection Reagent (11668019) was purchased from Invitrogen. .. CCK-8 Assay:Article Title: Infectious bronchitis virus accessory protein 3a induces renal inflammatory injury by activating the NLRP3 inflammasome via ER calcium mobilization and mitochondrial ROS production. Article Snippet: Lipofectamine 2000 Transfection Reagent (11668019) was purchased from Invitrogen. .. Western Blot:Article Title: Infectious bronchitis virus accessory protein 3a induces renal inflammatory injury by activating the NLRP3 inflammasome via ER calcium mobilization and mitochondrial ROS production. Article Snippet: Lipofectamine 2000 Transfection Reagent (11668019) was purchased from Invitrogen. .. Lactate Dehydrogenase Assay:Article Title: High‐Throughput Screening Reveals That CeeNU Acts as a New NLRP3 Inflammasome Inhibitor Article Snippet: Anti‐mouse IL‐1β, Human IL‐1β, IL‐6, and TNF‐α ELISA kits (CEK1788, CEK1731, CEK1785, CEK1783) were from Bioworld (USA). .. Cytotoxicity (LDH) assay kit ( C10007 ), |